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Fig. 3 | Molecular and Cellular Pediatrics

Fig. 3

From: Role of second-hand smoke (SHS)-induced proteostasis/autophagy impairment in pediatric lung diseases

Fig. 3

Schema showing novel mechanism for SHS-induced pediatric lung dysfunction. Exposure to SHS during early lung development (neonates/pediatric population) may potentiate initiation of chronic lung disease via oxidative stress mediated proteostasis/autophagy impairment and/or induction of HIF-1α levels. Therapeutic intervention using proteostasis/autophagy inducing drugs may mitigate the SHS exposure-mediated pathogenic changes initiating pediatric lung diseases such as bronchopulmonary dysplasia (BPD) or adult COPD-emphysema-like childhood condition

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